15.3 The Heart’s Wiring

Time To Read

6–8 minutes

Date Last Modified

Age 44


Echcardiogram, ECG,

1

Sharp, Pleuritic, Positional Chest Pain With a Friction Rub

2

Structurally Normal Chambers and Valves on Echo

3

Diffuse, Concave ST Changes Across Nearly All Leads

4

Normal S1 and S2 With a Superimposed Friction Rub

5

A Steady, Un-Tetanizable Rhythm Throughout the Flare

6

An Inflamed Serous Sac — Friction Rub Plus Small Effusion

7

Recurrent Pericarditis as FMF Serositis Reaching the Heart

The ER team ran an ECG within minutes of Stina’s arrival. To her, the tracing was a jagged ribbon; to the physician, it was a second confirmation. The ST-segment changes were spread diffusely across nearly all the leads instead of clustering in the few leads that watch one coronary artery’s territory. That pattern told them the irritation was wrapped around the whole heart — a sac problem, not a blocked-pipe problem — and it matched the rub they had already heard.

Every heartbeat begins as electricity. The sinoatrial (SA) node in the right atrium fires spontaneously — the heart’s natural pacemaker — and the impulse spreads across both atria to make them contract. It then reaches the atrioventricular (AV) node, which deliberately delays the signal so the atria can finish topping off the ventricles, then races down the bundle of His, the right and left bundle branches, and out through the Purkinje fibers, so the ventricles contract from the apex up in a coordinated squeeze. The ECG records this as the P wave (atrial depolarization), the QRS complex (ventricular depolarization), and the T wave (ventricular repolarization). Because pericarditis irritates the surface of the entire heart evenly, its ECG changes are diffuse; because a heart attack starves one arterial territory, its changes are focal. Reading where the changes sit on the heart is how Stina’s clinicians separated her inflamed sac from a clogged artery.

For Stina, the ECG is the wiring telling the same story the stethoscope told. Because pericarditis irritates the entire surface of the heart evenly, its ST changes are diffuse, spread across nearly all the leads; because a heart attack starves one artery’s territory, its changes are focal. Her tracing was diffuse — a whole-surface problem, not a blocked pipe — and it matched the friction rub the team had already heard. Two independent readings of the same inflamed sac, arriving at the same answer.

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