20.5 Acid-Base Balance & the Bicarbonate Buffer System

Time To Read

4–6 minutes

Date Last Modified

Age 44


Partially compensated renal aciosis

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Edema + Low Albumin + ‘Low’ Calcium 

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Edema Localized to a Plasma → Interstitium Shift

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Hypoalbuminemia → Low Oncotic Pressure

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A ‘Low’ Calcium That Is Really Lost Albumin

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A Low-Bicarbonate Drift = Metabolic Acidosis

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Renal Metabolic Acidosis, Only Partly Compensated

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Amyloid-damaged glomerulus

Blood pH is held in an astonishingly narrow band, around 7.35 to 7.45, because the proteins and enzymes that run the body only work within it. And here is the catch: you manufacture hydrogen ions all day long — from protein breakdown, from lactic acid, from ketones — without the matching base to cancel them. Your default drift is always toward acid; life is chemically a constant fight not to go acidic. The first line of defense is buffering, and the most important buffer in blood is the bicarbonate system.

Carbon dioxide plus water makes carbonic acid, which splits into bicarbonate and a hydrogen ion — and the whole reaction runs both directions, freely and constantly. That reversibility is the trick. When acid rises, bicarbonate mops up the extra hydrogen ions and the reaction shifts to make more CO2, which the lungs exhale; when acid falls, it shifts the other way. Bicarbonate is the body’s acid sponge, and CO2 is the volatile acid it can literally breathe off.

Two organs command the two ends of this reaction, and that is what makes it powerful. The lungs control CO2 — breathe faster and you blow off CO2, pulling toward less acid (a callback to the respiratory module, where CO2 first appeared as an acid in disguise). The kidneys control bicarbonate and fixed-acid excretion — reclaiming filtered bicarbonate, generating new bicarbonate, and secreting hydrogen ions into the urine. A disturbance is named for where it starts: respiratory if the problem is CO2, metabolic if it’s bicarbonate or fixed acid. Stina’s low-bicarbonate drift is a metabolic acidosis.

List of terms